AKT (v-akt murine thymoma viral oncogene homolog 1) and N-Ras (neuroblastoma ras viral oncogene homolog) coactivation in the mouse liver promotes rapid carcinogenesis by way of mTOR (mammalian target of rapamycin complex 1), FOXM1 (forkhead box M1)/SKP2, and c-Myc pathways.

AKT (v-akt murine thymoma viral oncogene homolog 1) and N-Ras (neuroblastoma ras viral oncogene homolog) coactivation in the mouse liver promotes rapid carcinogenesis by way of mTOR (mammalian target of rapamycin complex 1), FOXM1 (forkhead box M1)/SKP2, and c-Myc pathways / Ho, C; Wang, C; Mattu, S; Destefanis, G; Ladu, S; Delogu, S; Armbruster, J; Fan, L; Lee, Sa; Jiang, L; Dombrowski, F; Evert, M; Chen, X; Calvisi, Diego Francesco. - In: HEPATOLOGY. - ISSN 0270-9139. - 55:(2012), pp. 833-845.